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Hypotensive and hypertensive effects have been postulated? Demodex are active with stress, sun, harsh skincare products, heat, warm spicy foods which are triggers for a flare. Demodex trigger flushing and redness through the action of increased blood flow, and damage blood vessels, induces an inflammatory immune response with free radicals which further damages sebaceous gland and blood vessel right underneath the skin.
Demodex adults, larvae and eggs live in the bottom of the sebaceous gland lobules, when they come out of the glands, their effective pincers eat the sebum and create lipase which is an irritant and they can produce inflammatory chemicals like cytokines-key to facial, ocular, and scalp Rosacea.
Scalp Rosacea symptoms consist of chronic redness, burning sensations, cystic pustules and papules, hair loss.
The lobules look like cauliflower in the sebaceous gland histology, in normal skin there is only one Demodex mite, but in Rosacea skin it is like a balloon of Demodex mites at the end of the lobule.
The pincers chew on the sebaceous gland to get out and promote inflammatory reactions. They go out at night and go into other sebaceous glands. Demodex are active at night; people feel an itchy, crawly feeling as the Demodex go from one sebaceous gland to another.
The redness from an acne breakout is from inflammation not blood flow. Demodex larvae feeds off the sebum, waste product is lipase, inflammatory, as well as cytokines.
Chemokines are responsible for the immune response reaction- they send out dozen different signals to tell the local facial immune system that something is wrong and the immune system sends out macrophages, neutrophils, release oxidative bursts of free radicals that kill everything, and Leukotrienes.
This causes mast cell reactions-mast cells contain 1-part histamine and 12-parts Leukotrienes.
How does Rosacea affect ocular health? Efficacy of ivermectin againstDirofilaria immitis larvae in dogs 31, 60, and 90 days after injection. Ivermectin kills by interfering with nervous system and muscle function, in particular by enhancing inhibitory neurotransmission.
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